Cholesterol Deficiency – new genetic defect transmitted to Polish Holstein-Friesian cattle
نویسندگان
چکیده
منابع مشابه
Genetic Association Analysis of Paratuberculosis Forms in Holstein-Friesian Cattle
A genetic susceptibility to Mycobacterium avium subsp. paratuberculosis (MAP) infections in ruminants has been longtime suspected to exist. Recently, natural infections in cattle have been reclassified into latent and patent forms based on histopathological findings and their associations with immunological and microbiological variables. This study aims to explore whether these newly defined ph...
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The study was carried out at Agarfa ATVET College dairy farm to evaluate the reproductive performance of Holstein Friesian× Arsi and Holstein Friesian ×Boran cattle. For the study, records compiled from 1983 to 2012 at the Agarfa dairy farm were used as original data. The effects of breed, bloodlines, season and parity on the reproductive traits were evaluated. Data were analyzed using the gene...
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BACKGROUND Cholesterol deficiency (CD), a newly identified autosomal recessive genetic defect in Holstein cattle, is associated with clinical signs of diarrhea, failure to thrive, and hypocholesterolemia. HYPOTHESIS/OBJECTIVES The objective is to describe the clinicopathological phenotype of affected Holstein cattle homozygous for the causative apolipoprotein B gene (APOB) mutation. ANIMALS...
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Two types of cytochrome P450 2D14 (CYP2D14) gene deletions (GD1 and GD2) in Japanese Black cattle has been reported by us. In this study, we determined the frequency of cytochrome P450 2D14 (CYP2D14) genetic polymorphisms in 48 Japanese Black (JB) and 48 Holstein-Friesian (HF) cattle using polymerase chain reaction (PCR)-direct sequencing. Two types of CYP2D14 gene deletion variants, GD1 and GD...
متن کاملIdentification and prevalence of a genetic defect that causes leukocyte adhesion deficiency in Holstein cattle.
Two point mutations were identified within the gene encoding bovine CD18 in a Holstein calf afflicted with leukocyte adhesion deficiency (LAD). One mutation causes an aspartic acid to glycine substitution at amino acid 128 (D128G) in the highly conserved extracellular region of this adhesion glycoprotein, a region where several mutations have been found to cause human LAD. The other mutation is...
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ژورنال
عنوان ژورنال: Polish Journal of Veterinary Sciences
سال: 2016
ISSN: 2300-2557
DOI: 10.1515/pjvs-2016-0110